aiqbench analysis
Seq2Cause introduces a unified framework that leverages a single frozen autoregressive backbone to solve four distinct causal discovery regimes within discrete event sequences. By establishing a duality between next-token prediction error and causal identification bounds, the method scales to large vocabularies (up to 8,000 types) without requiring task-specific retraining. This approach effectively bridges the gap between predictive modeling and rigorous causal inference for complex systems like vehicle diagnostics and genomic data.
seq2cause: One Autoregressive Backbone, Four Causal Discovery Tasks in Event Sequences
arXiv:2609.31801v1 Announce Type: new Abstract: Complex systems such as vehicles, patients, or genomes emit discrete event sequences whose operative question is causal, not predictive: which events cause which other events, and which cause higher-level outcomes such as failures or diseases? This question decomposes along two axes -- dependency type (event $\to$ event vs.\ event $\to$ outcome) and causal scope (single sequence vs.\ population) -- yielding four structurally distinct regimes with different identifiability conditions. No existing method addresses more than one, because all assume multi-stream structure with low vocabulary, and none scales beyond a few hundred event types. We present \textsc{Seq2Cause}, a unified framework that resolves all four regimes through a single shared primitive: a pretrained autoregressive model repurposed as an amortized conditional independence testing engine requiring no task-specific retraining. We establish a prediction--causality duality: the model's excess cross-entropy simultaneously bounds causal identification error across all four regimes, so that every improvement in next-token prediction tightens causal guarantees for free. On nonlinear SCMs (vocabularies up to $8{,}000$ types) and real-world vehicle diagnostic logs ($29$K event types, $474$ failure outcomes), \textsc{Seq2Cause} is the first method to populate all four regimes at scale with a single frozen backbone. Existing methods are either inapplicable, inaccurate, or computationally intractable in this setting.
Abstract:Complex systems such as vehicles, patients, or genomes emit discrete event sequences whose operative question is causal, not predictive: which events cause which other events, and which cause higher-level outcomes such as failures or diseases? This question decomposes along two axes -- dependency type (event $\to$ event vs.\ event $\to$ outcome) and causal scope (single sequence vs.\ population) -- yielding four structurally distinct regimes with different identifiability conditions. No existing method addresses more than one, because all assume multi-stream structure with low vocabulary, and none scales beyond a few hundred event types. We present \textsc{Seq2Cause}, a unified framework that resolves all four regimes through a single shared primitive: a pretrained autoregressive model repurposed as an amortized conditional independence testing engine requiring no task-specific retraining. We establish a prediction--causality duality: the model's excess cross-entropy simultaneously bounds causal identification error across all four regimes, so that every improvement in next-token prediction tightens causal guarantees for free. On nonlinear SCMs (vocabularies up to $8{,}000$ types) and real-world vehicle diagnostic logs ($29$K event types, $474$ failure outcomes), \textsc{Seq2Cause} is the first method to populate all four regimes at scale with a single frozen backbone. Existing methods are either inapplicable, inaccurate, or computationally intractable in this setting.
Machine Learning (cs.LG)
arXiv:2609.31801 [cs.LG]
(or arXiv:2609.31801v1 [cs.LG] for this version)
https://doi.org/10.48550/arXiv.2609.31801 Focus to learn more arXiv-issued DOI via DataCite (pending registration)
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