Skip to content
aiqbench takeAI Research

aiqbench analysis

This research investigates how biologically inspired mechanisms, such as homeostasis and structural plasticity, can accelerate "grokking"—the delayed transition from memorization to generalization in neural networks. By augmenting multilayer perceptrons with these features, the study demonstrates that regulating neuron utilization significantly improves the emergence of robust, generalizable internal computations. These findings offer a promising pathway for optimizing training dynamics and reducing optimization time in future large language models.
Source coveringarXiv
What this isA model's reading of the article and related coverage, written for aiqbench. Not the publisher's words.

Biologically Inspired Mechanisms for Facilitating Grokking in Multilayer Perceptrons

arXiv2 min read

arXiv:2608.28184v1 Announce Type: new Abstract: Grokking is a delayed transition from memorization to generalization that is often accompanied by substantial reorganization of internal representations. This paper studies whether biologically inspired mechanisms, many of which are not commonly incorporated into artificial neural networks, can actively promote this transition by regulating hidden-layer computation at the levels of neuronal activity, response, and effective connectivity. We augment a multilayer perceptron with input gating, structural plasticity, gain modulation, threshold modulation, homeostasis, lateral inhibition, and activation decorrelation, and evaluate these mechanisms through systematic ablations on two established grokking benchmarks: sparse parity and noisy XOR classification. The results show that the mechanisms contribute unequally to generalization. Homeostasis provides the strongest and most consistent benefit, while structural sparsification emerges as the second major mechanism. The remaining biologically inspired mechanisms have smaller or less consistent effects in the present experiments. For both problems, the results support the common principle that explicit regulation of neuron utilization and effective connectivity can improve the emergence of generalizable internal computation. These findings motivate broader investigation of biologically inspired activity regulation and adaptive sparsification, including in large language models, where they may accelerate the development of generalizable representations and reduce the optimization time required for robust generalization.

Abstract:Grokking is a delayed transition from memorization to generalization that is often accompanied by substantial reorganization of internal representations. This paper studies whether biologically inspired mechanisms, many of which are not commonly incorporated into artificial neural networks, can actively promote this transition by regulating hidden-layer computation at the levels of neuronal activity, response, and effective connectivity. We augment a multilayer perceptron with input gating, structural plasticity, gain modulation, threshold modulation, homeostasis, lateral inhibition, and activation decorrelation, and evaluate these mechanisms through systematic ablations on two established grokking benchmarks: sparse parity and noisy XOR classification. The results show that the mechanisms contribute unequally to generalization. Homeostasis provides the strongest and most consistent benefit, while structural sparsification emerges as the second major mechanism. The remaining biologically inspired mechanisms have smaller or less consistent effects in the present experiments. For both problems, the results support the common principle that explicit regulation of neuron utilization and effective connectivity can improve the emergence of generalizable internal computation. These findings motivate broader investigation of biologically inspired activity regulation and adaptive sparsification, including in large language models, where they may accelerate the development of generalizable representations and reduce the optimization time required for robust generalization.

Machine Learning (cs.LG)

arXiv:2608.28184 [cs.LG]

(or arXiv:2608.28184v1 [cs.LG] for this version)

https://doi.org/10.48550/arXiv.2608.28184 Focus to learn more arXiv-issued DOI via DataCite (pending registration)

arXivLabs is a framework that allows collaborators to develop and share new arXiv features directly on our website.

Both individuals and organizations that work with arXivLabs have embraced and accepted our values of openness, community, excellence, and user data privacy. arXiv is committed to these values and only works with partners that adhere to them.

Have an idea for a project that will add value for arXiv's community? Learn more about arXivLabs.

Full text under the publisher's open licence · 292 words

Related coverage