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This research introduces a topological causal framework using persistent homology to detect distributional shape changes that traditional mean-based metrics like ATE and CATE miss. By formalizing a persistent-homology ignorability condition, the authors prove identifiability for these new estimands while clarifying mathematical nuances regarding mixtures over covariates. The approach offers a rigorous method for capturing treatment effects that alter the geometry of outcome laws without shifting their means.
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Beyond Means: Topological Causal Effects under Persistent-Homology Ignorability

arXiv3 min read

arXiv:2603.14169v2 Announce Type: replace-cross Abstract: Average treatment effects (ATE) and conditional average treatment effects (CATE) are foundational causal estimands, but they target changes in expected outcomes and can miss treatment-induced changes in the shape of outcome distributions. A canonical failure mode occurs when control outcomes are unimodal, treated outcomes become bimodal, and both distributions have the same mean. In such cases mean-based causal estimands are zero even though the geometry and topology of the outcome law change substantially. This paper develops a topological causal framework based on persistent homology. We formalize a persistent-homology ignorability condition, define topological analogues of CATE and ATE, and prove that these estimands are identifiable up to an explicit error bound under approximate topological ignorability. We also clarify a subtle but important point: a marginal persistence-diagram effect is not identified from conditional topological ignorability alone because persistent homology does not in general commute with mixtures over covariates. To preserve the original intuition while ensuring scientific correctness, we retain the marginal effect as a motivating quantity, but place the mathematically sound conditional estimands at the center of the theory. A synthetic experiment with mean-preserving topology change shows that mean-based causal estimands remain near zero while the proposed topological effect increases sharply and remains recoverable after adjustment for confounding.

Abstract:Average treatment effects (ATE) and conditional average treatment effects (CATE) are foundational causal estimands, but they target changes in expected outcomes and can miss treatment-induced changes in the shape of outcome distributions. A canonical failure mode occurs when control outcomes are unimodal, treated outcomes become bimodal, and both distributions have the same mean. In such cases mean-based causal estimands are zero even though the geometry and topology of the outcome law change substantially. This paper develops a topological causal framework based on persistent homology. We formalize a persistent-homology ignorability condition, define topological analogues of CATE and ATE, and prove that these estimands are identifiable up to an explicit error bound under approximate topological ignorability. We also clarify a subtle but important point: a marginal persistence-diagram effect is not identified from conditional topological ignorability alone because persistent homology does not in general commute with mixtures over covariates. To preserve the original intuition while ensuring scientific correctness, we retain the marginal effect as a motivating quantity, but place the mathematically sound conditional estimands at the center of the theory. A synthetic experiment with mean-preserving topology change shows that mean-based causal estimands remain near zero while the proposed topological effect increases sharply and remains recoverable after adjustment for confounding.

Methodology (stat.ME); Artificial Intelligence (cs.AI)

arXiv:2603.14169 [stat.ME]

(or arXiv:2603.14169v2 [stat.ME] for this version)

https://doi.org/10.48550/arXiv.2603.14169 Focus to learn more arXiv-issued DOI via DataCite

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Full text under the publisher's open licence · 300 words

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